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Body-Worn Sensors in Parkinson's Disease: Evaluating Their Acceptability to Patients.

These included deficiencies of tryptophan hydroxylase, catechol-O-methyltransferase, hydroxyindole methyltransferase the vescicular amine transporter, the pre-synaptic amine transporters, post synaptic receptors, GABA defects and other receptor disorders.

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The potential role of mitochondrial dysfunction and subsequent oxidative stress in the pathogenesis of PD was first suggested by the discovery that the administration of MPTP, a mitochondrial electron transport chain inhibitor, to rodents and primates, produced a phenotype similar to that observed in PD.

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In Parkinson disease, BDNF levels in the substantia nigra are reduced, and in animal models of Parkinson disease, BDNF provides a neuroprotective effect.

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